reptides / Noopept (omberacetam; GVS-111)

Noopept (omberacetam; GVS-111)

Noopept is omberacetam, a small molecule sold as a registered 10 mg tablet in Russia. In a 53-patient study of vascular or post-traumatic disorders, Noopept and piracetam groups improved, and some subgroup clinician ratings favored Noopept. The study had no placebo group, and two 20-person healthy-volunteer reports do not establish reliable cognitive enhancement.

Synthetic proline-glycine small molecule

  • Small molecule, not an amino-acid-chain peptide
  • Russian tablet evidence does not authenticate an online supplement
  • Healthy-person evidence consists of two reports with 20 participants each
Is it a peptide? Where is it registered? What did the 53-person study test? Does it improve healthy cognition? Are online products equivalent? What is known about PK and safety? How might it work?

Is Noopept actually a peptide?

No. Omberacetam is a defined chemical substance, N-phenylacetyl-L-prolylglycine ethyl ester. It contains a proline-glycine-derived scaffold, but FDA's substance record classifies it as a chemical and gives molecular formula C17H22N2O4 and molecular weight 318.37.

Noopept is a product name; omberacetam is the nonproprietary substance name. GVS-111 is a development synonym. A substance identity entry does not mean FDA approved the substance as a drug.

FDA GSRS · chemical identity

Official substance identity record

U.S. Food and Drug Administration, Global Substance Registration System

The record classifies omberacetam as a chemical, gives formula C17H22N2O4, molecular weight 318.37, CAS 157115-85-0, UNII 4QBJ98683M, and the systematic name N-phenylacetyl-L-prolylglycine ethyl ester.

  • Synonyms include Noopept and GVS-111.
  • A UNII identifies a substance; it is not an approval record.
Participants / model
Not applicable
Treatment
Not applicable
Study design
Official chemical-substance registry

The GSRS record establishes chemical identity; it does not establish efficacy or safety.

Read the original source

What is Noopept's current status in Russia and the United States?

Current Russian manufacturer materials describe Noopept as a registered 10 mg omberacetam tablet under ЛП-N(004920)-(РГ-RU), dated March 20, 2024. The registration claim comes from manufacturer materials; the cited evidence does not include a regulator record.

The Russian product indication covers adults with specified cognitive or emotional-lability disorders associated with traumatic brain injury, postconcussion syndrome, vascular cerebral insufficiency, encephalopathy, asthenia, or related loss of intellectual productivity. It is not a general authorization for healthy-person memory enhancement.

The cited Drugs@FDA records list no match for Noopept, omberacetam, or GVS-111 in brand or active-ingredient fields. A 2019 FDA warning letter told one U.S. seller that its named Noopept product was not a dietary supplement and was marketed as an unapproved new drug.

Official product PDF · indication, PK, safety

Manufacturer-hosted current product characteristics

OTCPharm, General Characteristics of the Medicinal Product, omberacetam 10 mg tablets

The Russian-language document gives the adult indication, 15-minute mean Tmax, 0.38-hour plasma half-life, named metabolites, contraindications, frequency-unknown adverse reactions, interaction wording, and a statement that withdrawal was not observed.

  • Contraindications include pregnancy, breastfeeding, age under 18, severe liver or kidney impairment, hypersensitivity, and lactose-related restrictions.
  • Frequency-unknown adverse reactions are allergic reaction and blood-pressure elevation in people with hypertension, especially severe hypertension.
Participants / model
Adults covered by the Russian product characteristics
Treatment
Noopept 10 mg omberacetam tablets
Follow-up
Product characteristics, not a single trial
Study design
Regulatory product document hosted by the manufacturer
Funding
OTCPharm

The manufacturer-hosted product characteristics provide the label statements; they do not supply the underlying human study results.

Read the original source
Manufacturer site · 2024 registration

Manufacturer product information

OTCPharm. Noopept product site.

The site identifies Noopept as omberacetam 10 mg tablets and displays registration ЛП-N(004920)-(РГ-RU), dated March 20, 2024.

Participants / model
Russian Noopept product
Treatment
Omberacetam 10 mg tablets
Study design
Manufacturer website
Funding
OTCPharm

Russian registration status is stated in manufacturer materials rather than a regulator record.

Read the original source
Drugs@FDA · no exact-name match

FDA drug records

Drugs@FDA records for Noopept, omberacetam, and GVS-111.

Drugs@FDA listed no matching brand-name or active-ingredient record for Noopept, omberacetam, or GVS-111.

Participants / model
Drugs@FDA application records
Treatment
Exact-name aliases Noopept, omberacetam, and GVS-111
Read the original source
FDA 2019 letter · named Noopept product

Official enforcement letter

U.S. Food and Drug Administration, February 5, 2019

FDA said the seller's Noopept product did not meet the statutory definition of a dietary supplement and that disease and structure/function claims made it an unapproved and misbranded new drug.

Participants / model
Products and marketing claims named in the letter
Treatment
Noopept sold by Peak Nootropics
Follow-up
Inspection and enforcement record issued in 2019
Study design
FDA enforcement document

The letter is seller- and claim-specific; it is not a clinical study or a blanket assay of all products.

Read the original source

What exactly did the 53-patient comparison show?

Both groups improved; the authors called the nootropic effects comparable, reported some subgroup clinician-rating advantages for Noopept, and reported undesirable effects 1.8-fold less often. The study compared Noopept with piracetam in patients with vascular or post-traumatic disorders, without placebo or healthy volunteers.

The full English translation describes 53 randomized patients: 31 assigned to Noopept 20 mg a day and 22 to piracetam 1,200 mg a day for 56 days. Thirty-seven patients had vascular-origin disorders and 16 had postconcussion syndrome. The paper does not describe how the random sequence was generated, allocation was concealed, or assessors were masked.

Forty-one patients completed. In the Noopept group, four vascular-disease patients stopped: one moved away, two had blood-pressure increases requiring antihypertensive treatment, and one reported no benefit. In the piracetam group, eight stopped across the two diagnostic groups, including adverse effects and lack of efficacy.

Small subgroups, missing placebo, unclear masking, and unequal attrition prevent the study from showing how much improvement came from either drug or whether Noopept helps healthy people.

53-patient comparison · no placebo or healthy group

Randomized active-comparator trial

Neznamov GG, Teleshova ES. Neuroscience and Behavioral Physiology. 2009;39(3):311-321

Among 53 patients assigned to Noopept or piracetam for 56 days, the authors reported cognitive and clinical improvement in both groups, comparable nootropic effects, stronger Noopept CGI results in some subgroups, and 1.8-fold fewer undesirable effects. Only 41 completed, there was no placebo, and the paper does not describe randomization or masking methods.

  • Noopept: 31 assigned, 20 mg/day. Piracetam: 22 assigned, 1,200 mg/day.
  • Two Noopept recipients stopped because blood pressure increased and required antihypertensive treatment.
Participants / model
37 patients with vascular-origin organic emotional-lability or asthenic disorder and 16 with postconcussion syndrome
Treatment
Noopept 20 mg/day versus piracetam 1,200 mg/day
Follow-up
56 days
Study design
Randomized comparative study with active comparator and no placebo; randomization and masking methods not reported

Small diagnostic subgroups and unequal attrition limit comparison.

Read the original source

What evidence exists in healthy volunteers?

Two small reports involved healthy volunteers, each with 20 people. One was a placebo-controlled conference abstract and the other was an uncontrolled Russian before-and-after report. Neither gives a dependable estimate of cognitive benefit.

What did the placebo-controlled abstract report?

Conference abstract P.4.E.052 describes 20 healthy men, 10 given oral DVD-111 at 20 mg a day for 13 days and 10 given placebo in a double-blind comparison. The authors reported favorable changes across spatial and logical thinking, attention, task speed, fine motor accuracy, reaction latency, and memory, with no side effects reported. The abstract gives no effect sizes, numerical outcome table, randomization method, multiplicity plan, or follow-up.

20-man placebo abstract · many unquantified outcomes

Double-blind placebo-controlled conference abstract

International Journal of Neuropsychopharmacology. Volume 5, Supplement 1, abstract P.4.E.052

The abstract describes 10 healthy men receiving oral DVD-111 at 20 mg/day for 13 days and 10 receiving placebo. It reports favorable changes across several cognitive and motor tasks and no side effects, but provides no numerical effect sizes, outcome table, randomization details, or multiplicity analysis.

Participants / model
20 healthy men
Treatment
DVD-111 20 mg/day, 10 participants, versus placebo, 10 participants
Follow-up
13 days
Study design
Double-blind placebo-controlled phase I conference abstract

The conference abstract provides no author metadata or numerical outcomes.

Read the original source
What did the Russian student report find?

A 2017 Russian conference-proceedings report measured the same 20 volunteers before and after one month of 20 mg a day. It reported short-term memory index rising from 40.0% to 62.2%, working memory from 29.3% to 36.2%, and long-term memory from 25.4% to 34.9%, with P<0.05 markers. Information-coding index and elementary-thinking speed did not change significantly. There was no placebo group, no masking, and no adverse-event reporting.

Russian n=20 report · uncontrolled before and after

Uncontrolled conference-proceedings report

Slobodenyuk TF. OPEN INNOVATION international conference proceedings. 2017:166-168

Twenty healthy volunteers took 10 mg twice daily for one month. The report found higher short-term, working, and long-term memory indices after treatment, but no significant change in information coding or elementary-thinking speed.

  • Short-term memory index was 40.0% before and 62.2% after treatment; working memory was 29.3% and 36.2%; long-term memory was 25.4% and 34.9%, with P<0.05 markers.
  • There was no concurrent control group, masking, or adverse-event account.
Participants / model
20 healthy volunteers described as students
Treatment
Noopept 10 mg twice daily
Follow-up
One month
Study design
Uncontrolled before-and-after cognitive testing

In this Russian report, "control" means each participant's pretreatment result, not a separate placebo group.

Read the original source

The controlled report is only an abstract with many tested outcomes, while the full Russian report is vulnerable to practice effects and expectation because it reused cognitive tests without a concurrent comparator. Neither is a replicated healthy-person efficacy program.

Does Russian tablet evidence apply to U.S. Noopept supplements?

No product equivalence has been shown. The clinical studies tested specified oral drug products, while a U.S. assay found undeclared drugs and inaccurate label quantities across a targeted sample of cognitive-enhancement supplements.

Researchers bought 10 products in 2019 after searching two supplement databases for five unapproved cognitive-enhancement drugs. Testing found up to four unapproved drugs in one product, nine of 12 declared quantities were inaccurate, and the maximum omberacetam amount was 40.6 mg per recommended serving. This targeted sample does not describe every product, but it shows that the name on a supplement label cannot establish identity, dose, or equivalence to the Russian 10 mg tablet.

U.S. supplement assay · undeclared drugs and wrong quantities

Targeted laboratory survey

Cohen PA, et al. Neurology: Clinical Practice. 2021;11(3):e303-e307

In 10 products bought in 2019, laboratory testing identified unapproved drugs including omberacetam, up to four unapproved drugs per product, and inaccurate quantities for nine of 12 drugs whose amounts were declared. The maximum omberacetam quantity was 40.6 ± 0.4 mg per recommended serving.

Participants / model
10 cognitive-enhancement supplements found through two U.S. supplement databases
Treatment
Product analysis, not human exposure
Follow-up
Products selected and purchased in 2019
Study design
Targeted LC-QTOF mass-spectrometry assay

This targeted sample demonstrates possible identity and quantity failures; it is not a prevalence estimate for every Noopept product.

Read the original source
FDA 2019 letter · named Noopept product

Official enforcement letter

U.S. Food and Drug Administration, February 5, 2019

FDA said the seller's Noopept product did not meet the statutory definition of a dietary supplement and that disease and structure/function claims made it an unapproved and misbranded new drug.

Participants / model
Products and marketing claims named in the letter
Treatment
Noopept sold by Peak Nootropics
Follow-up
Inspection and enforcement record issued in 2019
Study design
FDA enforcement document

The letter is seller- and claim-specific; it is not a clinical study or a blanket assay of all products.

Read the original source

How well are Noopept pharmacokinetics, adverse effects, and interactions defined?

The Russian label reports a mean 15-minute time to peak and a 0.38-hour parent-drug plasma half-life. Human data do not establish a dosing interval, cognitive-effect duration, or metabolite half-life.

A 2005 conference abstract describes a 10 mg oral test dose in patients with vascular or traumatic cognitive disorders. It reports that most reached maximum plasma concentration at 15 minutes, parent drug was detected through 45 minutes, and mean elimination half-time was 22.05 ± 14.37 minutes. It omits the human sample size and detailed concentration-time results. A separate 2004 abstract emphasizes considerable individual variability.

The current Russian label lists allergic reaction and blood-pressure elevation in people with hypertension, especially severe hypertension, with frequency unknown. It contraindicates use during pregnancy or breastfeeding, under age 18, with severe liver or kidney impairment, or with hypersensitivity. The tablets contain lactose.

The label says no interaction was established with alcohol, hypnotics, antihypertensives, or psychostimulants. That wording does not document a comprehensive interaction program. The 53-patient comparison reported worsened sleep, irritability, and increased blood pressure among Noopept recipients, including two discontinuations for blood-pressure increases.

The label also says withdrawal was not observed. The controlled trial lasted eight weeks, so this does not settle dependence, withdrawal after longer use, reproductive safety in humans, or chronic toxicity.

Official product PDF · indication, PK, safety

Manufacturer-hosted current product characteristics

OTCPharm, General Characteristics of the Medicinal Product, omberacetam 10 mg tablets

The Russian-language document gives the adult indication, 15-minute mean Tmax, 0.38-hour plasma half-life, named metabolites, contraindications, frequency-unknown adverse reactions, interaction wording, and a statement that withdrawal was not observed.

  • Contraindications include pregnancy, breastfeeding, age under 18, severe liver or kidney impairment, hypersensitivity, and lactose-related restrictions.
  • Frequency-unknown adverse reactions are allergic reaction and blood-pressure elevation in people with hypertension, especially severe hypertension.
Participants / model
Adults covered by the Russian product characteristics
Treatment
Noopept 10 mg omberacetam tablets
Follow-up
Product characteristics, not a single trial
Study design
Regulatory product document hosted by the manufacturer
Funding
OTCPharm

The manufacturer-hosted product characteristics provide the label statements; they do not supply the underlying human study results.

Read the original source
Human PK abstract · 10 mg oral test dose

Conference pharmacokinetic abstract

Bojko SS. European Neuropsychopharmacology. 2005;15:S222-S223

After a 10 mg oral test dose in patients with vascular or traumatic cognitive disorders, most reached peak parent-drug concentration at 15 minutes, plasma detection continued through 45 minutes, and mean elimination half-time was 22.05 ± 14.37 minutes.

Participants / model
Patients with cognitive disorders of vascular or traumatic origin; the abstract does not state the sample size
Treatment
Single oral 10 mg test dose, tablet or capsule formulations
Follow-up
Sampling reported through 45 minutes
Study design
HPLC pharmacokinetic conference abstract

The abstract omits the human sample size, detailed concentration-time data, and metabolite half-lives.

Read the original source
Comparative PK abstract · human variability

Indexed comparative pharmacokinetic abstract

Boiko SS, et al. Eksperimental'naia i Klinicheskaia Farmakologiia. 2004;67(1):40-43

The abstract reports slower elimination from rat to rabbit to human and considerable individual variability in humans. It does not give a numerical human half-life.

Participants / model
Rats, rabbits, and humans
Treatment
GVS-111 exposure; the abstract does not state the human dose
Follow-up
Single-dose pharmacokinetic work
Study design
Interspecies pharmacokinetic study reported in abstract form

The abstract reports no numerical human half-life.

Read the original source
53-patient comparison · no placebo or healthy group

Randomized active-comparator trial

Neznamov GG, Teleshova ES. Neuroscience and Behavioral Physiology. 2009;39(3):311-321

Among 53 patients assigned to Noopept or piracetam for 56 days, the authors reported cognitive and clinical improvement in both groups, comparable nootropic effects, stronger Noopept CGI results in some subgroups, and 1.8-fold fewer undesirable effects. Only 41 completed, there was no placebo, and the paper does not describe randomization or masking methods.

  • Noopept: 31 assigned, 20 mg/day. Piracetam: 22 assigned, 1,200 mg/day.
  • Two Noopept recipients stopped because blood pressure increased and required antihypertensive treatment.
Participants / model
37 patients with vascular-origin organic emotional-lability or asthenic disorder and 16 with postconcussion syndrome
Treatment
Noopept 20 mg/day versus piracetam 1,200 mg/day
Follow-up
56 days
Study design
Randomized comparative study with active comparator and no placebo; randomization and masking methods not reported

Small diagnostic subgroups and unequal attrition limit comparison.

Read the original source

Do NGF, BDNF, or cycloprolylglycine explain a human effect?

No human target mechanism has been established. The familiar NGF and BDNF explanation comes from animal experiments, and a later rat study under different conditions did not reproduce a hippocampal NGF or BDNF increase.

A Russian rat study reported higher hippocampal NGF and BDNF messenger RNA after acute and 28-day Noopept exposure. Another study divided 60 prepubertal male rats into six groups and found no spatial-learning or hippocampal NGF or BDNF difference after 14 days of 0.5 mg/kg intraperitoneal Noopept. The models, ages, disease conditions, routes, and doses differ, so the second experiment is context for uncertainty rather than a direct replication.

The label describes phenylacetic acid, phenylacetylproline, and cycloprolylglycine as metabolites and attributes activity to several pathways. Metabolite formation and animal gene-expression findings do not show which target, if any, mediates a clinical effect in people.

Rat study · hippocampal transcript changes

Preclinical animal study

Ostrovskaya RU, et al. Bulletin of Experimental Biology and Medicine. 2008

The rat experiment reported increased hippocampal NGF and BDNF messenger RNA after acute exposure and after a 28-day course.

Participants / model
Rats
Treatment
Noopept under experimental conditions
Follow-up
Acute and 28-day experiments
Study design
Preclinical gene-expression study

Gene-expression changes in rat hippocampus do not establish a human clinical mechanism.

Read the original source
Rat study · no learning or neurotrophin difference

Preclinical animal study

Karabulut S, et al. Behavioural Brain Research. 2019

Across six groups of 10 prepubertal male rats, the investigators found no difference in spatial learning or hippocampal NGF and BDNF after 14 days of intraperitoneal Noopept under the tested conditions.

Participants / model
60 prepubertal male rats, including healthy and diabetic groups
Treatment
Noopept 0.5 mg/kg intraperitoneally
Follow-up
14 days
Study design
Controlled preclinical animal experiment

Different age, disease models, route, and dose mean this is not a direct replication of the earlier rat study.

Read the original source
Official product PDF · indication, PK, safety

Manufacturer-hosted current product characteristics

OTCPharm, General Characteristics of the Medicinal Product, omberacetam 10 mg tablets

The Russian-language document gives the adult indication, 15-minute mean Tmax, 0.38-hour plasma half-life, named metabolites, contraindications, frequency-unknown adverse reactions, interaction wording, and a statement that withdrawal was not observed.

  • Contraindications include pregnancy, breastfeeding, age under 18, severe liver or kidney impairment, hypersensitivity, and lactose-related restrictions.
  • Frequency-unknown adverse reactions are allergic reaction and blood-pressure elevation in people with hypertension, especially severe hypertension.
Participants / model
Adults covered by the Russian product characteristics
Treatment
Noopept 10 mg omberacetam tablets
Follow-up
Product characteristics, not a single trial
Study design
Regulatory product document hosted by the manufacturer
Funding
OTCPharm

The manufacturer-hosted product characteristics provide the label statements; they do not supply the underlying human study results.

Read the original source

Studies and sources

FDA GSRS · chemical identity

Official substance identity record

U.S. Food and Drug Administration, Global Substance Registration System

The record classifies omberacetam as a chemical, gives formula C17H22N2O4, molecular weight 318.37, CAS 157115-85-0, UNII 4QBJ98683M, and the systematic name N-phenylacetyl-L-prolylglycine ethyl ester.

  • Synonyms include Noopept and GVS-111.
  • A UNII identifies a substance; it is not an approval record.
Participants / model
Not applicable
Treatment
Not applicable
Study design
Official chemical-substance registry

The GSRS record establishes chemical identity; it does not establish efficacy or safety.

Read the original source
Manufacturer site · 2024 registration

Manufacturer product information

OTCPharm. Noopept product site.

The site identifies Noopept as omberacetam 10 mg tablets and displays registration ЛП-N(004920)-(РГ-RU), dated March 20, 2024.

Participants / model
Russian Noopept product
Treatment
Omberacetam 10 mg tablets
Study design
Manufacturer website
Funding
OTCPharm

Russian registration status is stated in manufacturer materials rather than a regulator record.

Read the original source
Official product PDF · indication, PK, safety

Manufacturer-hosted current product characteristics

OTCPharm, General Characteristics of the Medicinal Product, omberacetam 10 mg tablets

The Russian-language document gives the adult indication, 15-minute mean Tmax, 0.38-hour plasma half-life, named metabolites, contraindications, frequency-unknown adverse reactions, interaction wording, and a statement that withdrawal was not observed.

  • Contraindications include pregnancy, breastfeeding, age under 18, severe liver or kidney impairment, hypersensitivity, and lactose-related restrictions.
  • Frequency-unknown adverse reactions are allergic reaction and blood-pressure elevation in people with hypertension, especially severe hypertension.
Participants / model
Adults covered by the Russian product characteristics
Treatment
Noopept 10 mg omberacetam tablets
Follow-up
Product characteristics, not a single trial
Study design
Regulatory product document hosted by the manufacturer
Funding
OTCPharm

The manufacturer-hosted product characteristics provide the label statements; they do not supply the underlying human study results.

Read the original source
Drugs@FDA · no exact-name match

FDA drug records

Drugs@FDA records for Noopept, omberacetam, and GVS-111.

Drugs@FDA listed no matching brand-name or active-ingredient record for Noopept, omberacetam, or GVS-111.

Participants / model
Drugs@FDA application records
Treatment
Exact-name aliases Noopept, omberacetam, and GVS-111
Read the original source
FDA 2019 letter · named Noopept product

Official enforcement letter

U.S. Food and Drug Administration, February 5, 2019

FDA said the seller's Noopept product did not meet the statutory definition of a dietary supplement and that disease and structure/function claims made it an unapproved and misbranded new drug.

Participants / model
Products and marketing claims named in the letter
Treatment
Noopept sold by Peak Nootropics
Follow-up
Inspection and enforcement record issued in 2019
Study design
FDA enforcement document

The letter is seller- and claim-specific; it is not a clinical study or a blanket assay of all products.

Read the original source
53-patient comparison · no placebo or healthy group

Randomized active-comparator trial

Neznamov GG, Teleshova ES. Neuroscience and Behavioral Physiology. 2009;39(3):311-321

Among 53 patients assigned to Noopept or piracetam for 56 days, the authors reported cognitive and clinical improvement in both groups, comparable nootropic effects, stronger Noopept CGI results in some subgroups, and 1.8-fold fewer undesirable effects. Only 41 completed, there was no placebo, and the paper does not describe randomization or masking methods.

  • Noopept: 31 assigned, 20 mg/day. Piracetam: 22 assigned, 1,200 mg/day.
  • Two Noopept recipients stopped because blood pressure increased and required antihypertensive treatment.
Participants / model
37 patients with vascular-origin organic emotional-lability or asthenic disorder and 16 with postconcussion syndrome
Treatment
Noopept 20 mg/day versus piracetam 1,200 mg/day
Follow-up
56 days
Study design
Randomized comparative study with active comparator and no placebo; randomization and masking methods not reported

Small diagnostic subgroups and unequal attrition limit comparison.

Read the original source
20-man placebo abstract · many unquantified outcomes

Double-blind placebo-controlled conference abstract

International Journal of Neuropsychopharmacology. Volume 5, Supplement 1, abstract P.4.E.052

The abstract describes 10 healthy men receiving oral DVD-111 at 20 mg/day for 13 days and 10 receiving placebo. It reports favorable changes across several cognitive and motor tasks and no side effects, but provides no numerical effect sizes, outcome table, randomization details, or multiplicity analysis.

Participants / model
20 healthy men
Treatment
DVD-111 20 mg/day, 10 participants, versus placebo, 10 participants
Follow-up
13 days
Study design
Double-blind placebo-controlled phase I conference abstract

The conference abstract provides no author metadata or numerical outcomes.

Read the original source
Russian n=20 report · uncontrolled before and after

Uncontrolled conference-proceedings report

Slobodenyuk TF. OPEN INNOVATION international conference proceedings. 2017:166-168

Twenty healthy volunteers took 10 mg twice daily for one month. The report found higher short-term, working, and long-term memory indices after treatment, but no significant change in information coding or elementary-thinking speed.

  • Short-term memory index was 40.0% before and 62.2% after treatment; working memory was 29.3% and 36.2%; long-term memory was 25.4% and 34.9%, with P<0.05 markers.
  • There was no concurrent control group, masking, or adverse-event account.
Participants / model
20 healthy volunteers described as students
Treatment
Noopept 10 mg twice daily
Follow-up
One month
Study design
Uncontrolled before-and-after cognitive testing

In this Russian report, "control" means each participant's pretreatment result, not a separate placebo group.

Read the original source
U.S. supplement assay · undeclared drugs and wrong quantities

Targeted laboratory survey

Cohen PA, et al. Neurology: Clinical Practice. 2021;11(3):e303-e307

In 10 products bought in 2019, laboratory testing identified unapproved drugs including omberacetam, up to four unapproved drugs per product, and inaccurate quantities for nine of 12 drugs whose amounts were declared. The maximum omberacetam quantity was 40.6 ± 0.4 mg per recommended serving.

Participants / model
10 cognitive-enhancement supplements found through two U.S. supplement databases
Treatment
Product analysis, not human exposure
Follow-up
Products selected and purchased in 2019
Study design
Targeted LC-QTOF mass-spectrometry assay

This targeted sample demonstrates possible identity and quantity failures; it is not a prevalence estimate for every Noopept product.

Read the original source
Human PK abstract · 10 mg oral test dose

Conference pharmacokinetic abstract

Bojko SS. European Neuropsychopharmacology. 2005;15:S222-S223

After a 10 mg oral test dose in patients with vascular or traumatic cognitive disorders, most reached peak parent-drug concentration at 15 minutes, plasma detection continued through 45 minutes, and mean elimination half-time was 22.05 ± 14.37 minutes.

Participants / model
Patients with cognitive disorders of vascular or traumatic origin; the abstract does not state the sample size
Treatment
Single oral 10 mg test dose, tablet or capsule formulations
Follow-up
Sampling reported through 45 minutes
Study design
HPLC pharmacokinetic conference abstract

The abstract omits the human sample size, detailed concentration-time data, and metabolite half-lives.

Read the original source
Comparative PK abstract · human variability

Indexed comparative pharmacokinetic abstract

Boiko SS, et al. Eksperimental'naia i Klinicheskaia Farmakologiia. 2004;67(1):40-43

The abstract reports slower elimination from rat to rabbit to human and considerable individual variability in humans. It does not give a numerical human half-life.

Participants / model
Rats, rabbits, and humans
Treatment
GVS-111 exposure; the abstract does not state the human dose
Follow-up
Single-dose pharmacokinetic work
Study design
Interspecies pharmacokinetic study reported in abstract form

The abstract reports no numerical human half-life.

Read the original source
Rat study · hippocampal transcript changes

Preclinical animal study

Ostrovskaya RU, et al. Bulletin of Experimental Biology and Medicine. 2008

The rat experiment reported increased hippocampal NGF and BDNF messenger RNA after acute exposure and after a 28-day course.

Participants / model
Rats
Treatment
Noopept under experimental conditions
Follow-up
Acute and 28-day experiments
Study design
Preclinical gene-expression study

Gene-expression changes in rat hippocampus do not establish a human clinical mechanism.

Read the original source
Rat study · no learning or neurotrophin difference

Preclinical animal study

Karabulut S, et al. Behavioural Brain Research. 2019

Across six groups of 10 prepubertal male rats, the investigators found no difference in spatial learning or hippocampal NGF and BDNF after 14 days of intraperitoneal Noopept under the tested conditions.

Participants / model
60 prepubertal male rats, including healthy and diabetic groups
Treatment
Noopept 0.5 mg/kg intraperitoneally
Follow-up
14 days
Study design
Controlled preclinical animal experiment

Different age, disease models, route, and dose mean this is not a direct replication of the earlier rat study.

Read the original source

Why is Noopept (omberacetam; GVS-111) in B tier?

The B tier reflects a registered-drug history and several human reports. Healthy-person evidence consists of two 20-person reports, and long-term safety, a human target mechanism, and equivalence with products sold online remain unestablished.

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